GDF-8 (Growth Differentiation Factor 8 Myostatin)
Purity: 85%
Specification: 1mg
Storage:2-8 degree centigrade refrigerator
Package: 10 Vials/Kit
Customized:accepted.
Country of Origin: China
GDF-8 (propeptide-Fc) is a myostatin inhibitor of muscle growth and development process. Myostatin is present in the healing process of muscles and its nature is to inhibit myogenesis (the process of formation of muscular tissue). Scientists have been studying a new method to enhance the repair and regeneration of both muscle and bone in certain injuries through a new recombinant myostatin propeptide.
GDF-8 works through the blocking of active myostatin made by a recombinant, myostatin propeptide has been able to improve muscle and bone regeneration. Myostatin inhibitors improve wound repair in cases of deep penetrant injuries on muscles and bones.
Some studies show that the lack of myostatin gives rise to impressive results in the increase of muscle mass both in humans and in animals. In bones specifically, the lack of myostatin increases its mineral density, resulting in an increase of the calcium levels of the organism. Calcium is stored in bones and it is necessary for the correct functionality of the heart, muscles, and nerves as well as for blood clotting.
While using this recombinant myostatin propeptide, it was found that collagen-rich fibrous tissue was plentiful in the injured area of the individual’s treated muscle, which has as a result the improvement of the regenerative capacity of the body.
Myostatin deficiency increases the ostegenic potential (formation of bone tissue) of bone marrow. There have been some studies related to the healing properties of stem cells originated in the bone marrow that has an auto regeneration capacity. This benefit can be promoted by blocking Myostatin on humans.
GDF-8 Peptideinhibitors are used on muscular dystrophy treatment in the attempt to accomplish the muscle regeneration. Myostatin propeptide enhances muscle regeneration, increases myofiber hypertrophy (an increase in size of skeletal muscle through a growth in size of its component cells) and decreases fibrosis (formation of excess fibrous connective tissue, which can obliterate the function of the affected limb) in muscle healing.
